Mold on Food: When to Cut and When to Toss
Quick Answer
For hard, dense foods like aged hard cheese, firm vegetables, and cured salami, you can cut 1 inch around and below the mold and safely eat the rest. For soft or moist foods (bread, soft cheese, yogurt, cooked leftovers, fruit, deli meat) discard the whole thing. The visible mold is the least of your worries. It's the invisible mycotoxins that spread deep into soft food that make the difference.
The Science
You’re about to make a sandwich and you find a small spot of mold on the bread. The rational brain immediately asks: does the whole loaf have to go, or can you just cut around it?
The answer depends on what the food is made of. Understanding why requires knowing what mold actually is and what it produces, both the visible parts you can see and the invisible compounds that don’t follow the mold’s edges.
What Mold Actually Is
Mold is fungi. Unlike bacteria (single-celled organisms), molds are complex, multicellular organisms that grow in a characteristic structure.
The visible mold you see on food is only part of the organism. Mold consists of two main structures:
Hyphae (singular: hypha) are long, thread-like filaments that mold sends down into a food source to absorb nutrients. They’re the fungal equivalent of roots. They penetrate the food, growing through its interior. In a piece of bread or a soft peach, hyphae can extend a centimeter or more into the food from the surface before the visible mold appears. By the time you see mold on top of bread, the hyphae are already well below the surface.
Spores are the reproductive structures that appear as the fuzzy, colored part you see. The color (green, black, white, blue) comes from the pigmentation of the spore-bearing structures. Spores are what spread through the air and start new mold colonies elsewhere.
The critical implication: the visible mold on the surface of food understates how far the mold has penetrated.
Mycotoxins: The Real Problem
Mold is gross but the mold itself isn’t the main safety hazard. Mycotoxins are.
Mycotoxins (from the Greek “mykes” for fungus and “toxicum” for poison) are chemical compounds that certain molds produce as secondary metabolites. They’re not essential to the mold’s growth. They’re more like defense mechanisms or byproducts of fungal metabolism under stress. And unlike mold cells themselves, mycotoxins are very small molecules that diffuse freely through food.
The three most significant mycotoxins in food:
Aflatoxin is produced by Aspergillus flavus and Aspergillus parasiticus, molds that grow on grains, nuts (especially peanuts and tree nuts), and dried fruits. Aflatoxin is a potent liver toxin and known human carcinogen. Chronic low-level exposure is associated with increased liver cancer risk. Aflatoxin B1 is the most potent naturally occurring carcinogen identified.
Ochratoxin A is produced by Aspergillus and Penicillium species. It’s found in cereals, coffee, dried fruits, and wine grapes. It accumulates in the kidneys and has nephrotoxic (kidney-damaging) and immunosuppressive effects.
Patulin is produced by Penicillium expansum and other molds that grow on apples, pears, and other pome fruits. It’s most concerning in apple juice, where the FDA monitors for it, because one moldy apple processed into juice can contaminate the whole batch.
Here’s the critical point: mycotoxins are not destroyed by cooking. They’re heat-stable molecules. Baking, boiling, microwaving, none of these eliminate mycotoxins already present in food. And they don’t stay in the mold. They diffuse through the food matrix.
The Cut-or-Toss Decision: A Science-Based Rule
The key variable is the food’s density and moisture content. These determine how far mycotoxins can diffuse and how deeply hyphae can penetrate.
In hard, dense, low-moisture foods, mold hyphae can’t penetrate deeply. The physical structure is too tight. Mycotoxins diffuse slowly through dense media. If you cut a generous margin (the USDA recommends 1 inch in all directions around and below the visible mold) you have a high probability of removing the mold and any associated toxins while leaving safe food.
In soft, moist foods, the food’s porous or high-moisture structure allows rapid hyphal penetration and fast mycotoxin diffusion. By the time you see surface mold, the interior of the food has already been infiltrated. There’s no safe margin you can cut that reliably removes all contamination.
| Food Category | Examples | Action |
|---|---|---|
| Hard, aged cheeses | Parmesan, aged cheddar, pecorino, gouda | Cut 1 inch around and below, use the rest |
| Firm vegetables | Carrots, cabbage, bell peppers, hard squash | Cut 1 inch around and below, use the rest |
| Cured, dry meats | Dry salami, hard pepperoni | Cut 1 inch around and below, use the rest |
| Soft cheeses | Brie, ricotta, cottage cheese, fresh mozzarella | Discard entire item |
| Soft fruits | Strawberries, peaches, tomatoes, grapes | Discard entire item |
| Bread and baked goods | Any bread, muffins, pastry | Discard entire item |
| Cooked leftovers | Casseroles, cooked rice, pasta | Discard entire item |
| Deli meat | Sliced turkey, ham, roast beef | Discard entire item |
| Yogurt, sour cream | Any soft dairy | Discard entire item |
| Jams and jellies | Any preserve | Discard entire item |
Note that jams and jellies, despite being high in sugar and seemingly shelf-stable, should be discarded when moldy. Mold can produce mycotoxins in jams, and the visible mold floating on top has likely sent compounds into the liquid below.
Aflatoxin in the food supply: the regulatory picture
Aflatoxin is present at low levels in many foods in the US food supply: peanuts, corn, tree nuts, and sometimes dried fruits. Regulatory agencies monitor for it because of its carcinogenicity.
The FDA sets action levels for aflatoxin:
- 20 parts per billion (ppb) for food intended for human consumption
- 0.5 ppb for milk (aflatoxin M1, which appears in milk from cows that ate contaminated feed)
Food at or above these levels is subject to seizure and destruction.
These limits exist because complete elimination of aflatoxin from the food supply isn’t achievable. Aflatoxin-producing molds grow in the field, before harvest, under conditions that aren’t fully controllable. Drought stress in particular promotes Aspergillus growth on corn and peanuts. The regulatory framework accepts low background levels while setting limits on what’s permitted for sale.
For consumers, this means the relevant exposure to aflatoxin isn’t usually a moldy piece of visible food. It’s background exposure in products that passed regulatory testing. The acute risk from visible mold on high-risk foods (peanuts, corn) is still real, but chronic background exposure is monitored at the national level.
Commercially produced peanut butter has some of the most thorough aflatoxin monitoring in the food system, because peanuts are a high-risk crop and peanut butter is consumed in large quantities. Testing is routine.
The Molds That Are Supposed to Be There
Blue cheese, brie, camembert, and certain other aged cheeses have intentionally introduced mold as a defining feature.
Blue cheese uses Penicillium roqueforti. The blue-green veins are hyphae running through the cheese interior. This mold produces some mycotoxins under certain conditions, but studies of well-made blue cheese consistently find either no detectable mycotoxins or levels far below any concern threshold. The conditions in properly made blue cheese (acidity, salt content, competition with other organisms) are not conducive to mycotoxin production.
Brie and camembert use Penicillium camemberti. The white rind is a mat of mold. Again, properly made under controlled conditions, this mold doesn’t produce harmful mycotoxins at concerning levels.
These designated molds are safe. The risk is when a different mold (one you didn’t invite) appears on the cheese. Green fuzzy mold on brie that isn’t part of the expected white rind is a contamination event. Discard it, even though you might be tempted to think “it’s just cheese mold.”
Preventing Mold in Your Kitchen
Mold needs moisture, moderate temperature, and a food source. Managing those conditions slows mold growth.
Refrigerate promptly. Mold grows slowly at refrigerator temperatures. Foods left at room temperature give mold much better growth conditions. This connects directly to the temperature danger zone. While the danger zone concept is primarily about bacteria, mold also grows faster in warmer conditions.
Keep produce dry. Water on the surface of produce accelerates mold growth. Pat berries dry before refrigerating. Don’t wash produce until you’re ready to use it.
Airflow matters. Mold spreads by spores in the air and by direct contact. Proper airflow in the refrigerator and pantry reduces the buildup of mold-favorable humidity. Don’t pack produce too tightly.
Check regularly. One moldy berry or piece of produce touching others accelerates mold spread through physical contact and spore transfer. Regular inspection catches mold early.
Understand your expiration dates. Products past their date that show mold are done. Products at or before their date that show mold are also done. The date doesn’t protect against mold.
The decision framework isn’t complicated once you internalize the key principle: with mold, what you see is not all you get. The visible fuzzy spot is the tip of the iceberg. In soft foods, the rest of the iceberg is already in the food.
What This Means for You
When you see mold on soft or moist food, don't cut around it. Discard the whole item. For hard cheeses and firm vegetables, cut a generous 1-inch margin around and below the mold and use the rest. Never smell mold directly. Spores inhaled in large quantities can cause respiratory irritation. Refrigerate promptly and keep produce dry to slow mold growth.
References
- USDA. Molds on Food: Are They Dangerous? Food Safety and Inspection Service.
- WHO. Mycotoxins. World Health Organization fact sheet.
- Alshannaq A, Yu JH. (2017). Occurrence, Toxicity, and Analysis of Major Mycotoxins in Food. International Journal of Environmental Research and Public Health. 14(6):632.
- Ostry V, Malir F, Toman J, Grosse Y. (2017). Mycotoxins as human carcinogens. Food and Chemical Toxicology. 99:187-200.
- Marin S, Ramos AJ, Cano-Sancho G, Sanchis V. (2013). Mycotoxins: Occurrence, toxicology, and exposure assessment. Food and Chemical Toxicology. 60:218-237.
- FDA. Aflatoxins: Protecting You from Aflatoxin Contamination. U.S. Food and Drug Administration.